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Ketofur 200 Solution *inj – 100 ml

Ketofur 200 (ketoprofen - 150 + ceftiofur hydrochloride - 50) (Ketofen analog)
Ceftiofur belongs to the group of third-generation cephalosporins. It is active against gram-positive and gram-negative bacteria, including strains producing beta-lactamases, and some anaerobic bacteria: Escherichia coli, Pasteurella haemolytica, Pasteurella multocida, Haemophilus Somnus, Actinobacillus pleuropneumoniae, Haemophilus parasuisocismellis, Salisuochemella parasuisis, Salisemis paraphuisis, Salis hemophilis paraleguis, Salmonis parasiusis, Pleisuoccephis Vulgaris, , Staphylococcus spp., Actynomyces pyogenes, Salmonella typhimurium, Streptococcus agalactiae, S. dysgalactiae, S. bovis, Klebsiella spp., Citrobacter spp., Enterobacter spp., Bacillus spp., Proteus spp., Poromomeromomium fungomomerumoma bomomerumomacteromumum ) The mechanism of action of the antibiotic is to suppress the synthesis of the bacterial cell wall. Ketoprofen - NSAIDs based on propionic acid. It has analgesic, anti-inflammatory and antipyretic effects. The mechanism of action is associated with inhibition of prostaglandin synthesis at the level of cyclooxygenase. In addition, ketoprofen inhibits lipoxygenase, has anti-bradykinin activity, stabilizes lysosomal membranes, and causes a significant inhibition of neutrophil activity. Ketoprofen is a dual inhibitor of inflammation, blocking the action of cyclooxygenase and lipoxygenase and, thus, preventing the formation of prostaglandins and leukotrienes. Ketoprofen is a painkiller that affects the central and peripheral nervous systems. Its effect is the direct suppression of the action of bradykinin, vasodilators and pain mediators.  

Ketofur 100 Solution – 100 ml

$38.69
Ketofur 100 (ketoprofen+ceftiofur hydrochloride)
Ketoprofen - NSAIDs based on propionic acid. It has analgesic, anti-inflammatory and antipyretic effects. The mechanism of action is associated with inhibition of prostaglandin synthesis at the level of cyclooxygenase. In addition, ketoprofen inhibits lipoxygenase, has anti-bradykinin activity, stabilizes lysosomal membranes, and causes a significant inhibition of neutrophil activity. Ketoprofen is a dual inhibitor of inflammation, blocking the action of cyclooxygenase and lipoxygenase and, thus, preventing the formation of prostaglandins and leukotrienes. Ketoprofen is a painkiller that affects the central and peripheral nervous systems. Its effect is the direct suppression of the action of bradykinin, vasodilators and pain mediators. After parenteral administration, ceftiofur is rapidly absorbed into the injection site and penetrates into all tissues of the animal. Its maximum plasma concentration is observed after an hour, which is maintained at a therapeutic level for up to 24 hours, depending on the type of animal. Ceftiofur in the body is rapidly metabolized to form desfuroilceftiofur, which has activity equivalent to ceftiofur against bacteria. This active metabolite binds back to plasma proteins and accumulates in the focus of infection, while its activity does not decrease in the presence of necrotic tissues. Withdrawal of the antibiotic occurs mainly with urine and feces.